Search results for "Muscle attachment"

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The muscleblind gene participates in the organization of Z-bands and epidermal attachments of Drosophila muscles and is regulated by Dmef2.

1998

We report the embryonic phenotype of muscleblind (mbl), a recently described Drosophila gene involved in terminal differentiation of adult ommatidia. mbl is a nuclear protein expressed late in the embryo in pharyngeal, visceral, and somatic muscles, the ventral nerve cord, and the larval photoreceptor system. All three mbl alleles studied exhibit a lethal phenotype and die as stage 17 embryos or first instar larvae. These larvae are partially paralyzed, show a characteristically contracted abdomen, and lack striation of muscles. Our analysis of the somatic musculature shows that the pattern of muscles is established correctly, and they form morphologically normal synapses. Ultrastructural a…

Central Nervous SystemSomatic cellMuscle Fibers SkeletalNeuromuscular JunctionMuscle ProteinsGenes InsectBiologymuscle attachmentsmuscleblindMesodermTendonsEctodermAnimalsDrosophila ProteinsConnectinRNA MessengerNuclear proteinMuscle SkeletalMolecular BiologyZ-bandsCell NucleusEpidermis (botany)MyogenesisMEF2 Transcription FactorsDrosophila.Gene Expression Regulation DevelopmentalNuclear ProteinsEmbryoCell DifferentiationCell BiologyAnatomybacterial infections and mycosesEmbryonic stem cellPhenotypeCell biologyDNA-Binding ProteinsMyogenic Regulatory FactorsVentral nerve cordMutationInsect ProteinsDrosophilaPhotoreceptor Cells InvertebratemyogenesisDevelopmental BiologyTranscription FactorsDevelopmental biology
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Terminal tendon cell differentiation requires the glide/gcm complex.

2004

International audience; Locomotion relies on stable attachment of muscle fibres to their target sites, a process that allows for muscle contraction to generate movement. Here, we show that glide/gcm and glide2/gcm2, the fly glial cell determinants, are expressed in a subpopulation of embryonic tendon cells and required for their terminal differentiation. By using loss-of-function approaches, we show that in the absence of both genes, muscle attachment to tendon cells is altered, even though the molecular cascade induced by stripe, the tendon cell determinant, is normal. Moreover, we show that glide/gcm activates a new tendon cell gene independently of stripe. Finally, we show that segment p…

[SDV]Life Sciences [q-bio]Cellglide/gcmBiologyMotor ActivityTendonsglide2/gcm203 medical and health sciencesTendon cellMuscle attachmentmedicineMuscle attachmentAnimalsDrosophila ProteinsRNA MessengerMolecular BiologyIn Situ Hybridization030304 developmental biology0303 health sciencesMuscles030302 biochemistry & molecular biologyNeuropeptidesTendon cell differentiationGene Expression Regulation DevelopmentalCell DifferentiationEpistasis GeneticAnatomyTendon cell differentiationEmbryonic stem cellCell biologyTendonDNA-Binding ProteinsMicroscopy ElectronDrosophila melanogasterSegment polarity genemedicine.anatomical_structureEpidermal CellsOrgan SpecificityTrans-ActivatorsDrosophilamedicine.symptomEpidermisLocomotionDevelopmental BiologyMuscle contractionProtein BindingSignal TransductionTranscription FactorsDevelopment (Cambridge, England)
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